Researchers have found that using pod-based e-cigarettes daily can elevate inflammatory markers across multiple organ systems.
The scientists saw that e-cigarettes caused a marked increase in inflammation within the brain’s reward pathways and that theseeffects also varied depending on e-cigarette flavor.
The study also suggested that e-cigarettes may influence how organs respond to infections such as SARS-CoV-2.
All kombucha has a bit of alcohol in it due to the sugar fermenting with the yeast. Commercial brands that have less than 0.5% by volume in it can be sold as “non-alcoholic.” Any more than that and the government considers it to be booze. Fermentation time, temperature, and the way the drink is stored all play roles in how strong it becomes. Some kombucha continues to ferment even in the bottle
Some reports link the homemade variety to stomachaches, dizziness, nausea, infections, and allergic reactions. The risk is high when people brew it in unclean conditions. That makes it easy to taint during fermentation. Brewing or storing it in glazed ceramic pots has a link to lead poisoning, too.
Stay away from this drink if you have a weakened immune system or a long-term health condition -- especially liver, kidney, or lung disease. Don’t drink it if you’re pregnant. It’s not for young children either. But if you’re a healthy adult, the store-bought kind that’s pasteurized is fine in moderation -- but not more than 12 ounces per day. Read the nutrition label, though, because the sugar and calories can vary quite a bit by brand.
It’s important to always keep kombucha in the refrigerator, even before you drink it. If you leave a capped bottle at room temperature for a while, the carbonation in it could build up -- and you might get a surprise splash when you open the bottle. The cold of the fridge also slows the fermentation.
Abstract - Aims: This study was designed to assess links between lifetime levels of marijuana use and accelerated epigenetic aging.
Design: Prospective longitudinal study, following participants annually from age 13 to age 30.
Findings: Lifetime marijuana use predicted accelerated epigenetic aging, with effects remaining even after covarying cell counts, demographic factors and chronological age (β's = 0.32 & 0.27, p's < 0.001, 95% CI's = 0.21-0.43 & 0.16-0.39 for DNAmGrimAge and DunedinPoAm, respectively). Predictions remained after accounting for cigarette smoking (β's = 0.25 & 0.21, respectively, p's < 0.001, 95% CI's = 0.14-0.37 & 0.09-0.32 for DNAmGrimAge and DunedinPoAm, respectively). A dose-response effect was observed and there was also evidence that effects were dependent upon recency of use. Effects of marijuana use appeared to be fully mediated by hypomethylation of a site linked to effects of hydrocarbon inhalation (cg05575921).
Conclusions: Marijuana use predicted epigenetic changes linked to accelerated aging, with evidence suggesting that effects may be primarily due to hydrocarbon inhalation among marijuana smokers. Further research is warranted to explore mechanisms underlying this linkage.
New study shows that those who had substance use disorder (SUD) symptoms when they were 18 years are more likely to have SUD symptoms in adulthood, more likely to use prescription drugs and more likely to have prescription drug misuse compared to those who had no SUD symptoms at 18 years. The risks are higher with those who had severe SUD symptoms when they were 18 years. (Dalgarno Institute – This is further vital evidence of the need to focus on Demand Reduction in for the emerging generation. Also see Protective Factor Number One in Drug Use Prevention Science)