Dean Whitlock refutes a claim that no one dies of marijuana overdose, in a section of his article, published by Vermont Digger, October 16, 2017.
“No One Has Died of an Overdose”
This remains the most outrageous claim of the pro-legalization movement. It is not only dangerously misleading, it is a slap in the face to the families who have lost children, spouses and parents.
Everyone admits that people are dying in traffic crashes because of stoned drivers, and that some people have died in butane hash oil explosions, but too many people are turning a blind eye to the other deaths caused by what can only be called an overdose.
Tachycardia – a racing heart – is a common, well-known side effect of using marijuana. So is increased blood pressure. A growing body of evidence, here and in other countries, is revealing that marijuana has caused previously overlooked deaths through heart attack and stroke. In Colorado last fall, an 11-month-old infant brought to the ER after being exposed to marijuana died from an inflamed heart muscle (myocarditis) caused by the exposure.
Marijuana can also overwhelm the emotional centers of the brain causing paranoia, delusions, and acute psychosis. The National Academy of Sciences (NAS) report released last January states, “There is substantial evidence of a statistical association between cannabis use and the development of schizophrenia or other psychoses, with the highest risk among the most frequent users.” (The Health Effects of Cannabis and Cannabinoids, Chapter Highlights NAS 2017)
Emergency rooms in Colorado reported a 44 percent increase in marijuana-related visits between 2012 and 2014. Many of these were cases of acute psychosis, particularly in young men, who had to be restrained to keep from harming themselves or others.
Author: Mark Gold, MD
Ground-breaking research on the hippocampus and ECs has enhanced our knowledge of the role of molecular mechanisms necessary to process lipid EC messengers. It has been established that circuitry between the hippocampus and prefrontal cortex is disrupted by the introduction of exogenous cannabinoid and thus is correlated with impairments in behavior (Kucewicz et al., 2011). Cannabinoids may disrupt cortical function by desynchronizing neuronal communication across cortical domains.
Why Does This Matter?
The deleterious effects of cannabinoids on learning and memory in humans are well documented via observational studies and neuropsychological testing of memory and cognitive abilities. The picture is becoming clearer.
Cannabinoid agonists (marijuana and cannabinoid synthetics) impair long-term potentiation in the hippocampus, resulting in cognitive impairments including attention deficits, poor executive functioning, memory impairment and disruption of temporal perception. Our UF students often described their marijuana smoking as causing ADD or learning problems that required morning cigarettes and psychostimulants to reverse. But they regularly failed, and marijuana succeeded in changing their career path and life trajectory from math, science, medicine or engineering to less rigorous and demanding majors and subsequent careers.
From Executive Summary
Section 4 – Emergency Department and Hospital Marijuana-Related Admissions:
The yearly rate of emergency department visits related to marijuana increased 35 percent after the legalization of recreational marijuana (2011-2012 vs. 2013-2015).
Number of hospitalizations related to marijuana:
2011 – 6,305
2012 – 6,715
2013 – 8,272
2014 – 11,439
Jan-Sept 2015 – 10,901
The yearly number of marijuana-related hospitalizations increased 72 percent after the legalization of recreational marijuana (2009-2012 vs. 2013-2015).
By Steve Doughty PUBLISHED: 5 October 2017
Cannabis users are more likely to commit violent crime, pioneering research has shown.
It warned those who smoke the drug regularly run an increased risk of using violence against others.
The project is the first to demonstrate that cannabis is not only linked with violent crime but is the cause.
Violent incidents monitored by the study based on the lives of more than 1,100 American psychiatric patients included assaults, attacks with weapons and rapes.
Researchers said that cannabis causes violence and they found no evidence that the link is the other way round – i.e. that violent people are more likely to use cannabis.
There was no support, they added, for theories put forward by campaigners anxious to free the drug from the taint of links with crime. The academics said the effect of cannabis use was clear and not diminished by other factors such as patients who were heavy drinkers of alcohol.
The study comes after a series of American states have decriminalised cannabis – despite it being stronger and more potent than the hash smoked by hippies in the Sixties – or made it available for medical use.
To conclude, our findings are relevant as they aid to shed light on the cannabis–violence association that has been less extensively studied amid psychiatric patients, in whom cannabis use is twice as prevalent in contrast to the general population. Compared to prior studies, we employed a prospective design to precisely examine the association between the continuation of cannabis use and violence. Our results are particularly relevant and may have clinical and violence risk management implications as we exposed that the persistency of cannabis use across different time waves was associated with an increased risk of violence in a large sample of patients recently discharged from acute psychiatric facilities.